詳細介紹
ZAP-70抗原 Zeta鏈相關蛋白激酶70
廣州健侖生物科技有限公司
該抗原是一種酪氨酸蛋白激酶,在T細胞和NK細胞中表達,參與T細胞受體的激活。該基因突變會使人類患重癥復合性免疫缺陷綜合癥(SCID)。該抗原還與慢性淋巴細胞白血?。–LL)病人的Ig重鏈基因突變狀態(tài)、病情進展和病人預后有關。所以該抗體可用于CLL的預后研究。
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ZAP-70抗原 Zeta鏈相關蛋白激酶70
【產(chǎn)品介紹】
細胞定位:細胞漿
克隆號:2F3.2
同型:IgG
適用組織:石蠟/冰凍
陽性對照:慢性淋巴細胞白血病/扁桃體
抗原修復:熱修復(EDTA)
抗體孵育時間:30-60min
產(chǎn)品編號 | 抗體名稱 | 克隆型別 |
OB234 | T-bet(T盒子轉錄因子) | MRQ-46 |
OB235 | TCL1試劑(T細胞淋巴瘤1) | MRQ-7 |
OB236 | TdT(末端脫氧核苷酸轉移酶) | polyclonal |
OB237 | TFE3試劑(轉錄因子E3) | MRQ-37 |
OB238 | Thyroglobulin(甲狀腺球蛋白) | DAK-Tg6 |
OB239 | Thyroglobulin(甲狀腺球蛋白) | 2H11+6E1 |
OB240 | TIA-1(T細胞胞漿內抗原) | 2G9A10F5 |
OB241 | Topo Ⅱ α(拓撲異構酶Ⅱα) | SD50 |
OB242 | TPO(甲狀腺過氧化物酶) | AC25 |
OB243 | TS(胸苷酸合成酶) | TS106 |
OB244 | TSH 甲狀腺刺激激素 | polyclonal |
OB245 | TTF-1(甲狀腺轉錄因子1) | 8G7G3/1 |
OB246 | TTF-1(甲狀腺轉錄因子1) | SPT24 |
OB247 | Tyrosinase(酪氨酸酶) | T311 |
OB248 | Uroplakin III試劑(尿溶蛋白III) | SP73 |
OB249 | VEGF(血管內皮生長因子) | VG1 |
OB250 | VEGF(血管內皮生長因子) | polyclonal |
OB251 | Villin(絨毛蛋白) | CWWB1 |
OB252 | Vimentin(波形蛋白) | V9 |
OB253 | Vimentin(波形蛋白) | SP20 |
OB254 | WT1(腎母細胞瘤) | EP122 |
OB255 | ZAP-70試劑() | 2F3.2 |
ZAP-70抗原
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【公司名稱】 廣州健侖生物科技有限公司
【市場部】 歐
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【騰訊 】
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103室
近年來,iPS研究發(fā)表的數(shù)量和速度都很驚人,但這些研究還局限于檢測相對簡單的系統(tǒng)。對于復雜網(wǎng)絡引發(fā)的問題或疾病,就需要采用能描述網(wǎng)絡互作的定量技術或者轉基因動物模型。這些方面的進步將加深我們對重編程機制的理解,有助于早日實現(xiàn)人們對這一技術的期望。
美國亞利桑那州立大學宇宙學家Paul Davies和同事提出一個備受爭議的結論,癌癥是人體細胞試運轉“安全模式”,如同計算機系統(tǒng)在面臨外部威脅時試著保護自己那樣。
宇宙學家提出疾病起源新理論
該理論主要基于癌癥的進化,如果該理論正確,他們的模型提示,氧氣治療、感染病毒或細菌等一些非傳統(tǒng)療法可能尤其有效。
癌癥是人體細胞試運轉“安全模式”所產(chǎn)生的嗎,就像受損計算機系統(tǒng)在面臨外部威脅時試著保護自己那樣?這是美國亞利桑那州立大學宇宙學家Paul Davies和同事得出的結論,他們提出了一個備受爭議的癌癥起源新理論。該理論主要基于癌癥的進化根源。如果該理論正確,他們的模型提示,氧氣治療和感染病毒或細菌的一些非傳統(tǒng)療法可能尤其有效。
In recent years, the number and speed of iPS research publications have been staggering, but these studies have also been limited to the detection of relatively simple systems. For problems or diseases caused by complex networks, quantitative techniques or transgenic animal models that describe network interactions are needed. Advances in these areas will deepen our understanding of the reprogramming mechanism and help to bring about the early realization of this technology.
Paul Davies, an ASU cosmologist at the University of Arizona, and his colleagues came up with the most controversial conclusion that cancer is a "safe mode" of human cell testing, just as computer systems try to protect themselves in the face of external threats.
Cosmologists put forward a new theory of the origin of disease
The theory is based primarily on the evolution of cancer and if the theory is correct, their models suggest that some unconventional therapies such as oxygen therapy, infection with viruses or bacteria may be particularly effective.
Is cancer a "safe mode" of human cell commissioning, just as damaged computer systems try to protect themselves against external threats? This is the conclusion of cosmologist Paul Davies and colleagues at Arizona State University, They put forward a controversial new theory of cancer origin. The theory is based primarily on the evolutionary causes of cancer. If the theory is correct, their models suggest that oxygen therapy and some unconventional therapies that affect the virus or bacteria may be particularly effective.