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甲型流感病毒IgM酶聯(lián)免疫法檢測(cè)試劑盒
廣州健侖生物科技有限公司
廣州健侖長(zhǎng)期供應(yīng)各種PCR試劑盒,主要代理進(jìn)口和國(guó)產(chǎn)品牌的流行病毒PCR檢測(cè)試劑盒。例如:甲乙型流感病毒核酸檢測(cè)試劑盒、黃熱病毒核酸檢測(cè)試劑盒、諾如病毒核酸檢測(cè)試劑盒、登革病毒核酸檢測(cè)試劑盒、基孔肯雅病毒核酸檢測(cè)試劑盒、結(jié)核桿菌核酸病毒檢測(cè)試劑盒、孢疹病毒核算檢測(cè)試劑盒、西尼羅河病毒PCR檢測(cè)試劑盒、呼吸道合胞病毒核酸檢測(cè)試劑盒、冠狀病毒PCR檢測(cè)試劑盒等等。蟲媒體染病系列、呼吸道病原體系列、發(fā)熱伴出疹系列、消化道及食源感染系列。
廣州健侖長(zhǎng)期供應(yīng)各種流感檢測(cè)試劑,包括進(jìn)口和國(guó)產(chǎn)的品牌,主要包括日本富士瑞必歐、日本生研、美國(guó)BD、美國(guó)NovaBios、美國(guó)binaxNOW、英國(guó)clearview、凱必利、廣州創(chuàng)侖等主流品牌。
主要檢測(cè):甲型流感病毒檢測(cè)試劑、乙型流感病毒檢測(cè)試劑、甲乙型流感病毒檢測(cè)試劑、A+B流感病毒檢測(cè)試劑盒、流感病毒抗原快速檢測(cè)卡、流感病毒抗體快速檢測(cè)試劑盒、流感快速檢測(cè)試劑 c1c2。
甲型流感病毒IgM酶聯(lián)免疫法檢測(cè)試劑盒
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【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】 歐
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【騰訊 】
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室
(2)囊樣變性:好發(fā)于黃斑部及顳下鋸齒緣附近。邊緣清楚,呈圓形或類圓形,暗紅色。周邊部病灶呈網(wǎng)狀,為成簇而略顯高起的小紅點(diǎn)。黃斑囊樣變性呈蜂窩狀。
(3)霜樣變性:大多發(fā)生于赤道部和鋸齒緣附近,網(wǎng)膜表面可見(jiàn)到一些有細(xì)小白色或黃色顆粒覆蓋的區(qū)域,厚薄不均,如同覆蓋了一片白霜。此類變性可單獨(dú)出現(xiàn),也可和格子樣變性、囊樣變性同時(shí)出現(xiàn)。在赤道部融合成帶狀的稱蝸牛跡樣變性。
(4)鋪路石樣變性:好發(fā)于下方周邊網(wǎng)膜。表現(xiàn)為有色素邊緣的、淡黃色圓形或類圓形、境界清楚的多發(fā)性萎縮病灶,大大小小病灶列成一片呈鋪路石樣。病灶中央部脈絡(luò)膜毛細(xì)血管萎縮,露出脈絡(luò)膜大血管或白色鞏膜。
(5)視網(wǎng)膜加壓發(fā)白與不加壓發(fā)白:將鞏膜壓陷后,眼底的隆起部變?yōu)椴煌该鞯幕野咨?,稱為加壓發(fā)白。病情進(jìn)一步發(fā)展時(shí),不加壓也呈灰白色,稱為不加壓發(fā)白,其后緣有時(shí)形成一清晰的嵴,多見(jiàn)于上方周邊網(wǎng)膜,被認(rèn)為是玻璃體牽引的一個(gè)指征。
(6)干性視網(wǎng)膜縱向皺襞:皺襞自鋸齒緣的齒緣間向赤道部方向伸展,是過(guò)度生長(zhǎng)視網(wǎng)膜組織的折疊,皺襞后端受玻璃體牽引易發(fā)生裂孔。玻璃體為一透明狀膠體樣結(jié)構(gòu),充填于眼球內(nèi)后部4/5的空腔內(nèi),對(duì)視網(wǎng)膜神經(jīng)上皮層貼著于色素上皮層,起著支撐作用。玻璃體變性時(shí),必然發(fā)生玻璃體后脫離,此時(shí)玻璃體從視網(wǎng)膜分離,分離往往從眼球的后上部開始,繼而向前擴(kuò)展。隨著脫離范圍的擴(kuò)大,甚至僅留下粘連緊密的視盤處。此時(shí)眼睛的迅速運(yùn)動(dòng)將產(chǎn)生玻璃體的連續(xù)地旋轉(zhuǎn)擺動(dòng),使視盤處玻璃體視網(wǎng)膜粘連分離,即形成了玻璃體*脫離。玻璃體脫離時(shí),對(duì)其下視網(wǎng)膜有牽拉作用,或引起視網(wǎng)膜裂孔,或使以前已存在的裂孔活化,促進(jìn)了視網(wǎng)膜脫離的發(fā)生。玻璃體全脫離時(shí),僅在基底部與視網(wǎng)膜粘連,脫離的后部玻璃體以此為支點(diǎn)發(fā)生搖擺,在此處產(chǎn)生明顯的牽拉作用,而周邊網(wǎng)膜又是各種視網(wǎng)膜變性的好發(fā)區(qū)域,非常容易受牽拉造成視網(wǎng)膜裂孔,這也是為什么視網(wǎng)膜脫離時(shí)裂孔多位于周邊網(wǎng)膜的原因。
(2) cystic degeneration: occur in the macula and near the jagged edge of the temporal. Clear edges, round or oval, dark red. Peripheral lesions were reticular, clustered and slightly elevated red dot. Cystoid degeneration was honeycomb.
(3) frost-like degeneration: mostly occurs in the vicinity of the equator and the serrated edge, the surface of the omentum can be seen some small white or yellow particles covered area, uneven thickness, as covered with a white frost. Such degeneration can appear alone, but also and lattice-like degeneration, cystic degeneration appear at the same time. Symptomatic snail-like degeneration that merges into a ribbon at the equator.
(4) stone pavement degeneration: occur in the bottom of the surrounding omentum. Performance for the edge of the pigment, pale yellow round or round, multiple clear atrophy territory, large and small lesions were paved stone-like. Central choroidal capillaries atrophy, showing choroidal blood vessels or white sclera.
(5) retina pressure whitening and no pressure whitening: the sclera after the depression, the raised fundus becomes opaque gray, known as pressure whitening. Further development of the disease, no pressure is gray, known as no pressure whitening, the trailing edge sometimes form a clear crest, more common in the upper peripheral omentum, is considered an indication of vitreous traction.
(6) Longitudinal folds of dry retina: Folds from the edge of the serrated edge to the equatorial extension of the retinal tissue is over-grown folds, folds of the posterior end of the vitreous traction prone to fracture. The vitreous body is a transparent colloid-like structure, which is filled in the cavity of 4/5 in the posterior part of the eyeball, playing a supportive role in attaching the retinal neurocortical layer to the pigment epithelium. Vitreous degeneration, the inevitable vitreous detachment, when the vitreous detachment from the retina, the detachment often from the top of the eye after the start, and then move forward. With the extension of the scope of separation, and even leave only the close adhesion of the disc at. At this point the rapid eye movement will produce a continuous rotation of the vitreous swing, the disc at the vitreoretinal adhesions, that is, the formation of a complete detachment of the vitreous. Vitreous detachment, the traction on the lower retina, or cause retinal holes, or to activate the pre-existing hiatus, and promote the occurrence of retinal detachment. Vitreous full detachment, only the basal part of the retina and adhesion, detachment of the posterior vitreous body as a fulcrum swing, where there is a significant pulling effect, and the perineural retina is a variety of retinal degeneration prone areas, Very easy to pull caused by retinal tears, which is why the retinal detachment more than in the surrounding omentum reasons.